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Showing posts with label science. Show all posts
Showing posts with label science. Show all posts

Friday, November 16, 2012

Bird of Paradise (Paradisaeidae)


Bird of Paradise

 
Paradisaeidae


Photo: Ribbon-tailed bird of paradise on a branch
Divas of the avian world, elaborately feathered birds of paradise, like this ribbon-tailed species, practice elaborate courtship rituals.
Photograph by Robert Sisson

Saturday, November 10, 2012

Ring-Necked Pheasant


Ring-Necked Pheasant

 
Phasianus colchicus

Photo: A ring-necked pheasant warily crosses a gravel road
More commonly found on the ground, ring-necked pheasants can take rapidly to the air when startled.
Photograph by George F. Mobley

Sunday, November 4, 2012

Greater Rhea: Largest bird in South America


Greater Rhea

 
Rhea americana


Photo: Greater rhea
As the largest bird in South America, the flightless greater rhea stands at about 4 feet (1.2 meters) tall.
Photograph by Nicole Duplaix

Sunday, September 9, 2012

Raven


Raven

 
Corvus corax (Northern Raven)


Photo: A male raven preens in the warm spring sunshine
Object of myth and poetry, the common raven is an intelligent bird of prey.
Photograph by Michael S. Quinton

Friday, August 17, 2012

Golden Eagle


Golden Eagle

 
Aquila chrysaetos


Photo: Golden eagle with a mountain backdrop
The national bird of Mexico, golden eagles are North America's largest raptor.
Photograph by Joe McDonald/Animals Animals—Earth Scenes

Friday, August 10, 2012

Peregrine Falcon


Peregrine Falcon

 
Falco peregrinus


Photo: Peregrine falcon
Once an endangered species in the United States, North American peregrine falcon populations have made a great comeback due to bans on usage of DDT and similar pesticides.
Photograph by Michael Melford

Monday, August 6, 2012

GMOs bad effects on animals

GMOs cause animals to lose their ability to reproduce, Russian scientists discover

(NaturalNews) A study presented at the Days of Defense Against Environmental Hazards in Russia has unveiled once again the implicit dangers associated with the consumption of genetically-modified organisms (GMOs). According to Voice of Russia, scientists from the National Association for Gene Security and the Institute of Ecological and Evolutional Problems discovered that animals fed GMOs as part of their normal diet eventually develop the inability to reproduce.

A shocking revelation; the study is one of several that has identified a link between GMO consumption and infertility in recent years. And because it was conducted independently of the biotechnology industry, it came to far different conclusions than have most of the studies offered up for mainstream consumption in defense of GMOs.



Using hamsters as the test subjects, scientists observed that consumption of GM soybeans, which have never been a part of these creatures' normal diets, tended to slow sexual maturity and gradually eliminate the ability to reproduce. After several generations, hamsters consuming the "Frankensoy" eventually lost their innate ability to reproduce.

"We selected several groups of hamsters, kept them in pairs in cells and gave them ordinary food as always," said Dr. Alexei Surov, one of the authors of the study. "We did not add anything for one group but the other was fed with soya that contained no GM components, while the third group with some content of Genetically Modified Organisms and the fourth one with increased amount of GMO."

Dr. Surov and his team then monitored the various groups of cubs, including their rates of growth and birthing frequencies, and compared their findings to the cubs' diets. After several generations of cubs breeding with each other, the team observed that those cubs who ate GMOs as part of their diet eventually became completely sterile.

"[W]e noticed quite a serious effect when we selected new pairs from [the] cubs and continued to feed them as before," added Dr. Surov. "These pairs' growth rate was slower and reached their sexual maturity slowly. When we got some of their cubs, we formed the new pairs of the third generation. We failed to get cubs from these pairs, which were fed with GM foodstuffs. It was proved that these pairs lost their ability to give birth to their cubs."

Will widespread GMO consumption make future generations of humans infertile?

An Austrian study uncovered similar harm in mice that consumed GM corn. An immediate consequence among the mice eating the "Frankencorn" was that their offspring weighed less than normal, and overall litter size became smaller. After three or four generations, mice who came from GMO-eating parents and grandparents became completely infertile. (http://www.naturalnews.com)

Farmers across the U.S. have also reported infertility among pigs and cows that consume GM feed. And in India, an investigatory team observed infertility and other serious problems among buffalo that were consuming GM cottonseed.

Sources for this article include:

http://english.ruvr.ru/2010/04/16/6524765.html

http://www.naturalnews.com

http://www.huffingtonpost.com

Learn more: http://www.naturalnews.com/036710_GMO_animal_experiments_infertility.html#ixzz22pJ5yXuX

Thursday, July 26, 2012

Gorilla Youngsters


Gorilla Youngsters Seen Dismantling Poachers' Traps—A First

"Very confident" four-year-olds outsmart hunters and protect their clan.

gorillas picture: young gorillas dismantle poacher's snare
Wild gorillas Rwema and Dukore destroy a primitive snare in Rwanda earlier this week.
Photograph courtesy Dian Fossey Gorilla Fund
Ker Than
Published July 19, 2012
Just days after a poacher's snare had killed one of their own, two youngmountain gorillas worked together Tuesday to find and destroy traps in theirRwandan forest home, according to conservationists on the scene.
"This is absolutely the first time that we've seen juveniles doing that ... I don't know of any other reports in the world of juveniles destroying snares," saidVeronica Vecellio, gorilla program coordinator at the Dian Fossey Gorilla Fund'sKarisoke Research Center, located in the reserve where the event took place.
"We are the largest database and observer of wild gorillas ... so I would be very surprised if somebody else has seen that," Vecellio added.
Bush-meat hunters set thousands of rope-and-branch snares in Rwanda's Volcanoes National Park, where the mountain gorillas live. The traps are intended for antelope and other species but sometimes capture the apes.
Adults are generally strong enough to free themselves. Youngsters aren't always so lucky.
Just last week an ensnared infant named Ngwino, found too late by workers from Karisoke, died of snare-related wounds. Her shoulder had been dislocated during escape attempts, and gangrene had set in after the ropes cut deep into her leg.
The hunters, Vecellio said, seem to have no interest in the gorillas. Even small apes, which would be relatively easy to carry away for sale, are left to die.
All-Natural Arsenal
Poachers build the snares by tying a noose to a branch or a bamboo stalk, Vecellio explained.
Using the rope, they pull the branch downward, bending it. They then use a bent stick or rock to hold the noose to the ground, keeping the branch tense. A sprinkling of vegetation camouflages the noose.
When an animal budges the stick or rock, the branch springs upward, closing the noose around the prey. If the creature is light enough, it will actually be hoisted into the air.
(See National Geographic magazine mountain gorilla pictures.)
Rwema and Dukore Save the Day
Every day trackers from the Karisoke center comb the forest for snares, dismantling them to protect the endangered mountain gorillas, which the International Fund for Nature (IUCN) says face "a very high risk of extinction in the wild."
On Tuesday tracker John Ndayambaje spotted a trap very close to the Kuryama gorilla clan. He moved in to deactivate the snare, but a silverback named Vubu grunted, cautioning Ndayambaje to stay away, Vecellio said.
Suddenly two juveniles—Rwema, a male; and Dukore, a female; both about four years old—ran toward the trap.
As Ndayambaje and a few tourists watched, Rwema jumped on the bent tree branch and broke it, while Dukore freed the noose.
The pair then spied another snare nearby—one the tracker himself had missed—and raced for it. Joined by a third gorilla, a teenager named Tetero, Rwema and Dukore destroyed that trap as well.
Gorilla Tactics
The speed with which everything happened makes Vecellio, the gorilla program coordinator, think this wasn't the first time the young gorillas had outsmarted trappers.
"They were very confident," she said. "They saw what they had to do, they did it, and then they left."
Silverbacks in the Kuryama group have occasionally been caught in the snares, so Vecellio thinks the juveniles would have known the traps are dangerous.
"That's why they destroyed them," Vecellio said.
"Quite Ingenious"
Despite the unprecedented nature of the event, Vecellio said she wasn't surprised by the reports. "But," she said, "I'm always amazed and very proud when we can confirm that they are smart."
Veterinarian Mike Cranfield, executive director of the Mountain Gorilla Veterinary Project, also said he wasn't shocked by the news.
"Chimpanzees are always quoted as being the tool users, but I think, when the situation provides itself, gorillas are quite ingenious," he said.
Cranfield speculated that the gorillas may have learned how to destroy traps by watching the Karisoke center's trackers.
"If we could get more of them doing it, it would be great," he joked.
Karisoke's Vecellio, though, said actively instructing the apes would be against the center's ethos.
"No we can't teach them," she said. "We try as much as we can to not interfere with the gorillas. We don't want to affect their natural behavior."

Tuesday, July 24, 2012

Lobsters and climate change


Lobsters to Be Supersized by Climate Change?

A small lobster is pictured at left, next to a larger lobster at right.
American lobsters raised in more acidic waters grow much larger (above, right) than lobsters grown in less acidic conditions (left), a December 2009 study found.

The experiment, which mimicked rising atmospheric carbon dioxide (CO2) levels that are predicted for Earth's atmosphere, shows acidic seas can produce jumbo-size shelled marine species, including lobsters, crabs, and shrimp.
Photograph courtesy Justin Ries, UNC-Chapel Hill
Brian Handwerk
National Geographic News
Attention lobster lovers—climate change may supersize your favorite seafood. So says a new study showing that more acidic oceans, a consequence of rising atmospheric carbon dioxide (CO2) levels, can produce jumbo-size shelled marine species, including lobsters, crabs, and shrimp. It's unknown whether the animals' body mass also gets beefier along with their shells. (Learn aboutgreenhouse gases.)
But it's not good news for all sea creatures. In the experiment, other species—including oysters, scallops, and clams—suffered in more acidic waters because they had more trouble building their shells.
That's because rising CO2 levels boost the amount of carbon in the oceans, but reduce levels of the carbonate ion that marine organisms need to make their protective shells.
Study leader Justin Ries, of the University of North Carolina at Chapel Hill, and colleagues grew 18 species of shelled marine animals in conditions that mimicked the elevated CO2 levels predicted in Earth's atmosphere over the next two centuries.
The lobsters and other animals that thrived under higher CO2 levels during the experiment are better able to convert the inorganic carbon in seawater into a form they can use to produce shells.
Acid Tipping Point
The team also found that some species, such as calcifying algae, benefited from increased CO2 up to a point—and then quickly began to die.
This may illustrate a specific CO2 "tipping point," which scientists could identify and monitor in future studies, according to the report published in December'sGeology.
But even animals that grew larger in more acidic waters may ultimately suffer if an ocean food chain evolved over hundreds of millions of years is rapidly reorganized.
For instance clams—a main prey of crabs and lobsters—have "completely different" responses to rising CO2, Ries said.
"It's a scenario where the predator is getting stronger and the prey is getting weaker. The [clams] may not be able to sustain their populations and ultimately the predators will also be adversely impacted when the prey populations crash."
Ecological Engineers Threatened
The scenario also poses problems for "ecological engineers" such as coral, which at first had no response to the acidic water but then began to die, Ries said.
"If corals decline, like the experiment predicts, then all the organisms that need a coral reef to survive, like crabs and lobsters, are going to suffer, even though they are predicted to do well [in more acid conditions]," he said.
Likewise, the winners in acidic oceans may also see setbacks, he added.
For instance, if species are pouring more of their hard-earned energy into shell growth, other biological functions like reproduction may decline.

Thursday, July 19, 2012

Virus Discovered in Cultus Lake Sport Fish


Virus Discovered in Cultus Lake Sport Fish

A Simon Fraser University fish-population statistician, working in collaboration with non-government organization scientists, has uncovered evidence of a potentially deadly virus in a freshwater sport fish in B.C.
SFU professor Rick Routledge and Stan Proboszcz, a fisheries biologist at the Watershed Watch Salmon Society, have found evidence of the piscine reovirus (PRV) in cutthroat trout caught in Cultus Lake, in the Fraser Valley region of B.C. Tests conducted by, Fred Kibenge, a virology professor at the Atlantic Veterinary College in Prince Edward Island, found evidence of the virus in 13 of 15 sampled fish. Follow-up analyses further confirmed the virus's presence in these fish and identified their genetic sequencing as 99 per cent identical to Norwegian strains. The virus has been scientifically linked to heart and skeletal muscle inflammation (HSMI), a disease that has reportedly become widespread in Norwegian salmon farms and can kill up to 20 per cent of infected fish. Routledge believes this first ever discovery of PRV in a B.C. freshwater sport fish indicates the virus could be prevalent in B.C. Many Canadian scientists and interest groups are concerned that B.C. salmon farms pose a serious risk to wild Pacific salmon. Scientists in other countries have specifically raised concerns about the spread of PRV from farms to wild salmon. "If PRV has been found in a Cultus Lake sport fish it could be contributing to the failure of the lake's sockeye population to return in abundance," says Routledge. He notes the federal government-mandated Committee on the Status of Endangered Wildlife in Canada has listed the species as endangered. "The discovery of PRV in Cultus Lake's cutthroat trout also begs the question is it in other related species in the lake, such as rainbow trout, kokanee and Dolly Varden? This latest discovery could also mean that salmon and trout in any lake exposed to spawning salmon returning from the North Pacific must be considered at risk of infection."

Rick Routledge, an SFU fish-population statistician, says he has helped uncover a potentially deadly virus in a B.C. freshwater sport fish.

Earlier this year, SFU honorary degree recipient Alexandra Morton, an independent biologist who collaborates on fish research with Routledge, reported that lab tests had found PRV in Atlantic salmon sold in B.C. supermarkets. PRV is the second virus commonly associated with salmon farming that scientists say they have found in wild Pacific salmon and trout. Last fall, Routledge and Morton reported positive test results for the infectious salmon anaemia virus (ISAv) in sockeye salmon smolts. Scientists testifying at the Cohen Commission Inquiry — a federally commissioned investigation of the Fraser River's declining sockeye population — have brought forth highly contested evidence of ISAv in other wild salmon populations, including Cultus Lake sockeye. The inquiry is scheduled to release its findings this fall. "We discovered during the Cohen inquiry that pathogens are a major concern for B.C.'s salmon," says Craig Orr, executive director of Watershed Watch and an SFU graduate. "Our findings suggest we need to broaden our thinking and concerns for freshwater fish as well." "There are many examples of devastating impacts of introduced pathogens in human, mammal and marine populations," adds Routledge. "When small pox was introduced to North America it decimated the aboriginal population, which had not had any previous opportunity to build up a natural immunity to the disease. The potential for this to happen to B.C.'s highly valued Pacific salmon and trout populations must be taken seriously." Provided by Simon Fraser University 

Tuesday, July 17, 2012

Spider Venom Could Treat Muscular Dystrophy


Protein Found in Spider Venom Could Treat Muscular Dystrophy

ScienceDaily (July 16, 2012) — While Spider-Man is capturing the imagination of theatergoers, real-life spider men in Upstate New York are working intently to save a young boy's life.

UB researchers are developing a treatment for muscular dystrophy using a peptide found in the venom of a Chilean rose tarantula. (Credit: Image courtesy of University at Buffalo)
It all began in 2009, when Jeff Harvey, a stockbroker from the Buffalo suburbs, discovered that his grandson, JB, had Duchenne muscular dystrophy. The disease is fatal. It strikes only boys, causing their muscles to waste away.
Hoping to help his grandson, Harvey searched Google for promising muscular dystrophy treatments and, in a moment of serendipity, stumbled upon University at Buffalo scientist Frederick Sachs, PhD.
Sachs was a professor of physiology and biophysics who had been studying the medical benefits of venom. In the venom of the Chilean rose tarantula, he and his colleagues discovered a protein that held promise for keeping muscular dystrophy at bay. Specifically, the protein helped stop muscle cells from deteriorating.
Within months of getting in touch, Harvey and Sachs co-founded Tonus Therapeutics, a pharmaceutical company devoted to developing the protein as a drug. Though the treatment has yet to be tested in humans, it has helped dystrophic mice gain strength in preliminary experiments.
The therapy is not a cure. But if it works in humans, it could extend the lives of children like JB for years -- maybe even decades.
Success can't come quickly enough.
JB, now four, can't walk down the stairs alone. When he runs, he waddles. He receives physical therapy and takes steroids as a treatment. While playing tee ball one recent day, he confided to his grandfather, "When I grow up, I want to be a baseball player." It was a heartbreaking moment.
"Oh, I would be thrilled if you could be a baseball player," Harvey remembers replying. He's doing everything he can to make sure that JB -- and other boys like him -- can live out their dreams.
Story Source:
The above story is reprinted from materials provided byUniversity at Buffalo.
link: http://www.sciencedaily.com/releases/2012/07/120716142657.htm 

Sunday, July 15, 2012

Cloned horses

Cloned horses may compete in the 2012 London Olympics, while anti-doping testing for humans becomes more sophisticated

(NaturalNews) While performance enhancement drugs and techniques may be banned for humans, the gate has been opened for genetically modified horses to compete in the Olympic games.

In a surprising but not shocking development, the Federation Equestre Internationale has decided to overturn a ban previously placed on cloned equines.

According to The Chronicle of the Horse at their recently held sports forum it was said, "The FEI will not forbid participation of clones or their progenies in FEI competitions. The FEI will continue to monitor further research, especially with regard to equine welfare."



Whereas in 2007, the stance of the FEI was the polar opposite; "The competitive equestrian couple of horse and rider are both acknowledged as athletes by the FEI. The cloning of either with a view to competing at international level would be unacceptable to the FEI. The FEI opposes cloning for it goes against one of the FEI's basic objectives: to enable FEI athletes 'to compete in international events under fair and even conditions."

This announcement comes in light of the London 2012 Olympics being touted as having "the most sophisticated anti-doping operation in the history of the Games." According to CNN, for the first time in Olympics history, a private company will provide its facilities - $30 million state-of-the-art laboratory - to aide in the testing of samples from Olympic participants to detect illegal performance enhancements. This is being provided by pharmaceutical giant GlaxoSmithKline (GSK). According to GSK, the facilities will offer "enable expert analysts from King's (College, London) to independently operate a World Anti-Doping Agency (WADA) accredited laboratory during the Games."

Although the FEI announcement has come too late for cloned horses to participate in the London 2012 Olympics, the implications will be far reaching, placing pressure on other equestrian governing bodies to relax the generally held stance of not allowing physiologically altering performance enhancement to be used by any participants.

This action has set a precedent for all sports, and the broader implications of such a development may mean that the rules pertaining to human performance enhancement may be contested, and become relaxed amid pressure from those who advocate its use.

Currently, the world's biggest companies offering cloning are ViaTech in Texas and Cryozootech in Sonchamp, France.

They have both welcomed the news, and stated that their main aim is to allow the genes of top horses that have died or been gelded to be available again.

Sources for this article include:

http://www.chronofhorse.com

http://edition.cnn.com

http://www.gsk.com/media/london-2012/index.htm

About the author:
Simon Victor is a health health and fitness enthusiast who believes in being practical with healthy living.
his personal journey can be followed at http://veganonabudget.net

Learn more: http://www.naturalnews.com/036474_cloned_horses_Olympics_doping.html#ixzz20iGif4tP

Friday, July 13, 2012

Aggressiveness in dog


If Your Dog Is Aggressive, Maybe It Is in Pain

ScienceDaily (June 13, 2012) — Dogs can sometimes suffer sudden episodes of aggression without their owners understanding why. But, in many cases, the cause of these attacks can be pain that has never been diagnosed or treated. For the first time the study describes the characteristics of this irritability, which can make dogs violent and increase aggression in already conflictive individuals.

The pain produced by hip dysplasia is a key factor in the risk of large dogs becoming aggressive. (Credit: Alex E. Proimos)
There are many factors that explain aggression in dogs: the conditions of the mother during gestation, the handling of the puppy in the neonatal phase, the age at weaning, the experiences of the animal during the socialising phase, diet, exercise, genetics and learning techniques based on active punishment during adulthood. However, aggressive behaviour also arises from the presence of pathologies and pain in the dog.
Between 2010 and 2011 a team of researchers from the department of Animal and Food Science at the Autonomous University of Barcelona (UAB) in Spain analysed the aggression problems of 12 dogs (Giant schnauzer, Irish setter, Pit-bull, Dalmatian, two German shepherds, Neapolitan Mastiff, Shih-tzu, Bobtail, Catalan Sheepdog, Chow-chow and Doberman) who were brought to the UAB's Veterinary Hospital by their owners.
"All (eleven males and one female) were diagnosed as having aggression caused by pain. Out of the 12 studied, eight had suffered a hip dysplasia," as was outlined by Tomás Camps, lead author of the study that was published in the Journal of Veterinary Behavior and researcher at the Animal Nutrition and Welfare Service of the UAB.
Scientists identified the most frequent circumstances in which dogs were aggressive, the most characteristic positions, the most frequent target of attacks and if they were impulsive or, in other words, whether or not they showed signs before an imminent attack.
The study shows that dogs that were already aggressive for other reasons before the onset of pain attacked their owners more intensely and frequently when food was taken away, when they were moved from their rest area or when they were made to do something. Animals displayed aggression in the same situations (or nearly) as those that were already aggressive.
On the other hand, "dogs that had never been aggressive before the onset of pain began to behave in this way in situations were an attempt is made to control them," points out Camps.
In addition, the study shows that those dogs were more impulsive or, in other words, attacked with no prior warning in the form of growls, for instance. The research team states that "if the pet is handled when in pain, it will quickly act aggressively to avoid more discomfort without the owner being able to prevent it."
Diagnosing hip dysplasia
Canine hip dysplasia affects more than 40% of Golden Retrievers, Labradors and Rottweillers and is a hereditary and degenerative bone disorder that affects the joint connecting the hip and the femoral head. In general, it can affect any breed of large dog but is less frequent is small breeds.
The new study suggests that the pain produced by hip dysplasia is a key factor in the risk of large dogs becoming aggressive. But the problem begins when the dog experiences periods of little pain and the illness is not diagnosed on time.
The researchers have reassessed the importance of diagnosis and treatment of pain since, according to Camps, "it can cause aggression or worsen aggression problems."
Story Source:
The above story is reprinted from materials provided byPlataforma SINC, via AlphaGalileo.
Note: Materials may be edited for content and length. For further information, please contact the source cited above.

Journal Reference:
  1. Tomás Camps, Marta Amat, Valentina M. Mariotti, Susana Le Brech, Xavier Manteca. Pain-related aggression in dogs: 12 clinical casesJournal of Veterinary Behavior: Clinical Applications and Research, 2012; 7 (2): 99 DOI:10.1016/j.jveb.2011.08.002

Wednesday, July 11, 2012

Chickens and Cancer


Ordinary Chickens May Be Extraordinary in Fighting Cancer and Other Diseases

ScienceDaily (July 10, 2012) — The common barnyard chicken could provide some very un-common clues for fighting off diseases and might even offer new ways to attack cancer, according to a team of international researchers that includes a Texas A&M University professor.
James Womack, Distinguished Professor of Veterinary Pathobiology in the College of Veterinary Medicine & Biomedical Sciences, is co-author of a paper detailing the team's work that appears in the current issue ofPNAS (Proceedings of the National Academy of Sciences). Womack was a leader in the international effort to sequence the cattle genome in 2004.


Womack and the team, comprised mostly of scientists from the Seoul National University in Korea, examined 62 White Leghorn and 53 Cornish chickens for diversity in NK-lysin, an antibacterial substance that occurs naturally in animals and is used as a method of fighting off diseases.
They were able to obtain two genetic variations of NK-lysin and the results offered two unexpected shockers: both showed abilities to fight off bacterial infections and other diseases, while one showed it could successfully fight cancer cells as well.
"It took all of us by surprise," Womack says of the findings.
"One of the genetic variations shows it has the ability to fight against cancer cells much more aggressively than the other variation. We certainly were not looking at the cancer side of this, but there it was."
Womack says the team selected the two breeds because Cornish and White Leghorn chickens, found throughout most of the world, have relatively diverse genetic origins.
After conducting a DNA sequence of the chickens, the team found two variations of the genes that offered clues as to their protective ability to ward off infections.
"One form appears to be more potent in killing off cancer cells than the other, and that's the one that naturally caught our eye," Womack adds.
"This could lead to other steps to fight cancer or in developing ways to prevent certain infections or even diseases. It's another door that has been opened up. We are looking at similar studies right now to see if this is possible with cattle.
"The next step is to work with other animals and see if similar variants exist. We need to look for any genetic similarities to the chicken variants and then determine if these variants affect the health of the animal, but this is an exciting first step in this direction."
Story Source:
The above story is reprinted from materials provided byTexas A&M University, via Newswise.
Note: Materials may be edited for content and length. For further information, please contact the source cited above.

Journal Reference:
  1. Mi Ok Lee, Eun-Hee Kim, Hyun-Jun Jang, Mi Na Park, Hee-Jong Woo, Jae Yong Han, and James E. Womack. Effects of a single nucleotide polymorphism in the chicken NK-lysin gene on antimicrobial activity and cytotoxicity of cancer cellsPNAS, July 10, 2012 DOI:10.1073/pnas.1209161109

Wednesday, July 4, 2012

Motion Sensors for Horse Lameness


Motion Sensors Detect Horse Lameness Earlier Than Veterinarians

ScienceDaily (July 3, 2012) — The most common ailment to affect a horse is lameness. A University of Missouri equine veterinarian has developed a way to detect this problem using a motion detection system called the "Lameness Locator." Now, Kevin Keegan, a professor of equine surgery in the College of Veterinary Medicine at MU, has found that his Lameness Locator can detect lameness earlier than veterinarians using the traditional method of a subjective eye test.

Equine veterinarians have developed a way to detect lameness using a motion detection system called the "Lameness Locator." (Credit: Image courtesy of University of Missouri-Columbia)

The Lameness Locator, which is now in commercial use, places small sensors on the horse's head, right front limb and croup, near the tail. The sensors monitor and record the horse's torso movement while the horse is trotting. The recorded information is then transferred to a computer or mobile device and compared against databases recorded from the movement of healthy horses and other lame horses. The computer is then able to diagnose whether or not the horse is lame.
In a new study published in theEquine Veterinary Journal, Keegan and co-author Meghan McCracken, an equine surgery resident at MU, put special adjustable shoes on horses that temporarily induced symptoms of lameness. The horses were then monitored by the Lameness Locator as well as by a number of veterinarians using any lameness testing methods they wished. If no lameness was detected by either the veterinarians or the Lameness Locator, the special shoes were adjusted slightly to increase the symptoms of lameness. This process was repeated until both the Lameness Locator and the participating veterinarians properly identified in which leg of the horse the lameness was occurring. Keegan and McCracken found that the Lameness Locator was able to correctly identify lameness earlier than veterinarians using subjective eye test methods more than 58 percent of the time and more than 67 percent of the time when the lameness occurred in the hind legs of the horse. Keegan attributes this to the sensors' high sensitivity levels.
"There are two reasons why the Lameness Locator is better than the naked eye," Keegan said. "It samples motion at a higher frequency beyond the capability of the human eye and it removes the bias that frequently accompanies human subjective evaluation."
Because equine lameness may begin subtly and can range from a simple mild problem affecting a single limb to a more complicated one affecting multiple limbs, veterinarians and horse owners know that early detection is the key to successful outcomes.
"If veterinarians can detect lameness earlier, before it gets too bad, it makes treatment much easier," Keegan said. "Lameness often goes undetected or undiagnosed entirely, which can cause owners to retire horses earlier than needed, simply because they cannot figure out why the horses are unhealthy. The Lameness Locator should be able to help with that as well."

Story Source:
The above story is reprinted from materials provided byUniversity of Missouri-Columbia.
Note: Materials may be edited for content and length. For further information, please contact the source cited above.

Journal Reference:
  1. M. J. Mccracken, J. Kramer, K. G. Keegan, M. Lopes, D. A. Wilson, S. K. Reed, A. Lacarrubba, M. Rasch.Comparison of an inertial sensor system of lameness quantification with subjective lameness evaluation.Equine Veterinary Journal, 2012; DOI: 10.1111/j.2042-3306.2012.00571.x

http://www.sciencedaily.com/releases/2012/07/120703162620.htm

Tuesday, July 3, 2012

Animal microchips


Animal microchips linked to causing cancer


(NaturalNews) Many veterinarians recommend them, and most animal shelters require them. Identification microchips injected into the necks of cats and dogs are touted as useful in recovering lost pets because the devices store owner and medical information. But are they safe? A new lawsuit against Merck & Co., Inc., maker of the HomeAgain pet microchip, says they are not, noting that they can cause cancer to develop in pets.



Featured at www.ChipMeNot.org, a website launched to raise awareness about the harm caused to animals by microchips, the lawsuit alleges that Merck's HomeAgain pet microchip induces cancerous tumors in pets. According to the suit, the defendant's cat developed cancer after getting a chip implant, and according to reports, other animals have gotten cancer after getting chipped as well.

"Based on the alarming number of microchip-induced cancers we're discovering, I predict this lawsuit will be just the tip of the iceberg," said Dr. Katherine Albrecht, a consumer advocate and expert on side effects associated with implantable microchips. "Merck and organizations that advocate pet chipping should take this lawsuit seriously and start warning pet owners of the risk of microchip-induced cancer."

According to the U.S. Food and Drug Administration, potential health risks associated with implantable microchips include "adverse tissue reaction". Based on data from the British Small Animal Veterinary Association, this can include "swelling", "infection", "abscesses", and "tumors".

Albrecht presented a paper on the subject called "Microchip-Induced Tumors in Laboratory Rodents and Dogs: A Review of the Literature 1990-2006" (http://www.chipmenot.org/pdfs/P074.pdf) at the June conference of the Institute of Electrical and Electronics Engineers that documents the increasing number of animals being harmed by microchips. Currently, there is no repository of data on adverse events associated with microchips in the U.S., but Albrecht organization, CASPIAN, is filling that void by compiling such information and making it available to the public.

To learn more about the dangers of animal microchips, visit: www.ChipMeNot.org

Sources for this story include:

http://www.chipmenot.org/mercksued.htm

Learn more: http://www.naturalnews.com/030108_microchips_animals.html#ixzz1zccfhZOD